Hormone replacement therapy for men

Currently, in today’s markets, there are numerous hormone treatment drugs that are produced to cater for the majority of people. Even though they can have general makeups that can benefit many people, some facts should be considered. Every woman is unique in their own way. Women have different chemical make ups, different health conditions, and different medical needs. Using a general HRT method may not cater for all your medical needs. It is just logical that you might require a unique setting with different concentrations of hormones with extra or fewer factors than the general HRT medications. For this reason, many women are turning to custom compounded HRT which we expertly specialize in.

Deep vein thrombosis (DVT) is a condition in which a blood clot forms in the deep vein of the leg or pelvis. It affects approximately 1 in 1000 people. If it is not treated, the clot can travel in the blood and block the arteries in the lungs. This life-threatening condition is called a pulmonary embolism (PE) and occurs in approximately 3 to 4 per 10,000 people. The chances of getting a DVT can be increased if people have certain risk factors. These include previous clots, prolonged periods of immobility (such as travelling on aeroplanes or bed rest), cancer, exposure to oestrogens (pregnancy, oral contraceptives or hormone replacement therapy), trauma and blood disorders such as thrombophilia (abnormal blood clotting). A DVT is diagnosed through determining the risk factors and performing an ultrasound of the leg veins. If a DVT is confirmed, people are treated with an anticoagulant. This medicine prevents further clots from forming. Until recently, the drugs of choice were heparin, fondaparinux and vitamin K antagonists. However, these drugs can cause side effects and have limitations. Two further classes of novel oral anticoagulants have been developed: these are called direct thrombin inhibitors (DTI) and factor Xa inhibitors. There are particular reasons why oral DTIs and factor Xa inhibitors might now be better medicines to use. They can be given orally, they have a predictable effect, they do not require frequent monitoring or re-dosing and they have few known drug interactions. This review measures the effectiveness and safety of these new drugs with conventional treatment.

In premenopausal women the majority of estrogen produced by the body is estradiol (produced primarily in the ovaries), while in postmenopausal women estrone (produced in fat cells) is the type of estrogen present in the greatest amount; however, the body is able to convert one type of estrogen into another to a certain extent. Because of the limited research into potency, delivery methods and conversion of the various estrogens, a valid scientific understanding of compounded estrogen products has not been achieved. [21] Synthetic estradiol, taken orally, splits when absorbed in the gastrointestinal tract and delivers bioidentical estradiol to the bloodstream. [22]

Comparisons between orally administered pill and transdermal patch suggests that when estrogens are taken orally the risks of thrombophlebitis and pulmonary embolism are increased, an effect which is not seen with topical administration. Transdermal and transvaginal administration are not subject to first pass metabolism , and so lack the anabolic effects that oral therapy has on hepatic synthesis of Vitamin K dependent clotting factors . [28] This effect refers only to patches for post menopausal hormone replacement, which contain estradiol , not those used in oral contraceptive therapy, which contain ethinylestradiol . The latter is associated with an increased incidence of venous clot. [29] The WHI also showed an increased incidence arterial disease, namely stroke, in patients who began HRT after the age of 65, although this effect was not significantly present in those who began therapy during their fifth decade.

Hormone replacement therapy for men

hormone replacement therapy for men

Comparisons between orally administered pill and transdermal patch suggests that when estrogens are taken orally the risks of thrombophlebitis and pulmonary embolism are increased, an effect which is not seen with topical administration. Transdermal and transvaginal administration are not subject to first pass metabolism , and so lack the anabolic effects that oral therapy has on hepatic synthesis of Vitamin K dependent clotting factors . [28] This effect refers only to patches for post menopausal hormone replacement, which contain estradiol , not those used in oral contraceptive therapy, which contain ethinylestradiol . The latter is associated with an increased incidence of venous clot. [29] The WHI also showed an increased incidence arterial disease, namely stroke, in patients who began HRT after the age of 65, although this effect was not significantly present in those who began therapy during their fifth decade.

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